Bluestone Silk N Blood 15 ~REPACK~

Bluestone Silk N Blood 15

to investigate further the mechanism of the protective vascular endothelial and perivascular epithelial restoration on ipsc-derived msc-treated allografts, we examined the levels of tissue oxygenation and blood perfusion in d4 and d14 untreated control allografts, which had not been treated with ipsc-derived mscs and served as untreated allograft controls. our data showed that untreated control allografts received no significant improvement in tissue hypoxia or blood perfusion at d4 or d14 post transplantation (fig. 4 a, b), suggesting that the mechanism of protection must be working in the very early period after transplantation, and beyond this, it may be too late to reverse the tissue hypoxic and ischemic state and restore graft tissue hypoxia and blood perfusion. on the other hand, the findings showed that the untreated allografts were significantly in a phase of graft hypoxia and ischemia by d4 and d14 post transplantation, and this was consistent with earlier graft rejection.

in summary, we found that the cymerus ipsc-derived mscs migrated to the grafts of the recipients and accelerated recovery of tissue oxygenation and improved blood perfusion of syngrafts and the control allografts. the ipsc-derived mscs were able to reduce ischemic tissue injury and inflammatory immune responses and normalized the immune homeostasis between t regulatory cells and the effector t cells in the allografts. the effects of the ipsc-derived mscs were greater than those of the syngrafts, suggesting that the mscs have extended the limit of reviving the grafts during rejection. our findings might represent a significant step toward future clinical application of cymerus ipsc-derived mscs in treating alloimmune rejection in allotransplantation.

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